Chapter 2.4 · Neuroanatomy

Neurological Syndromes — INBDE Review

The pattern-recognition payoff: Brown-Séquard, ASA infarct, MCA/ACA/PCA strokes, brainstem lesions, cerebellar syndromes, and the aphasias. Each one tied back to the anatomy from 2.1–2.3. 11 board-style MCQs.

11 practice MCQsQuick-reference tableMnemonics + clinical pearlsFull distractor explanations
High-yield review

Concept summary & clinical relevance.

Quick-reference structure first, then nerve-by-nerve detail. Mnemonics in amber, clinical pearls in blue.

Neurological syndromes are how the INBDE actually tests neuroanatomy: a brief vignette plus a request to localize the lesion. This chapter is the pattern-recognition payoff for everything in 2.1–2.3 — vascular territories, brainstem nuclei, cerebellar laterality, and the aphasia patterns that get asked over and over.

Stroke syndromes by vascular territory
ArteryClassic deficitSide
MCA (dominant, usually left)Aphasia + face/arm hemiparesisContralateral motor; language deficits independent of side
MCA (non-dominant, usually right)Hemineglect + face/arm hemiparesisContralateral motor + ignores left side of space
ACALeg > arm weakness; abuliaContralateral
PCAHomonymous hemianopia (often macular sparing); rarely prosopagnosiaContralateral visual field
Brainstem cranial nerve syndromes
Brainstem levelCN deficitClassic finding
MidbrainCN III palsyPtosis, “down and out” eye, dilated pupil
PonsCN V and VII palsyLoss of facial sensation/mastication; facial droop
PonsCN VI palsyInability to abduct eye
MedullaCN IX, X, XII deficitsLoss of gag, hoarseness, tongue deviation; respiratory failure if severe
Spinal cord syndromes
SyndromePattern
Brown-Séquard (hemicord)Ipsi motor + ipsi vibration; contra pain/temp (1–2 levels below)
Anterior spinal artery (ASA) infarctBilateral motor + pain/temp loss; vibration spared
Posterior spinal artery (PSA) infarctVibration/proprioception loss in isolation
Central cord syndromeBilateral pain/temp loss; motor + vibration spared
Clinical pearl — Aphasia in the operatory
If a patient suddenly produces fluent but meaningless speech and can't follow commands, that's Wernicke's aphasia. If speech is halting and effortful but they understand you, that's Broca's. Either is a stroke until proven otherwise — call EMS, document time of onset (last known well), and don't wait to see if it resolves.
Clinical pearl — Cerebellar lesions are ipsilateral
Unlike the cortex (where deficits are contralateral), cerebellar lesions produce ipsilateral signs because the cerebellum's input/output crosses twice. A right cerebellar hemisphere lesion → right-sided intention tremor and dysmetria. Vermis lesions affect trunk and gait.
Clinical pearl — Stroke recognition matters in dental practice
Sudden facial droop, slurred speech, unilateral weakness, or vision change in a patient = call EMS immediately. Document the last-known-well time — that's the clock thrombolytic eligibility runs on. Don't try to “wait it out” between procedures.
Mnemonic — Stroke vascular territories
“MCA = Mouth, ACA = Ankle, PCA = Picture.” MCA = face/arm + language. ACA = leg. PCA = vision.
Mnemonic — Brown-Séquard triangle
“Ipsi motor + ipsi vibes, contra pain.” Hemicord lesion gives ipsilateral motor + vibration loss with contralateral pain/temperature loss.
Mnemonic — Aphasia in two phrases
Broca's = Broken speech. Wernicke's = Word salad. Comprehension intact in Broca's; impaired in Wernicke's.

Cortical stroke syndromes

  • MCA territory: face and arm hemiparesis with cortical signs. Dominant hemisphere → aphasia (Broca's, Wernicke's, or global). Non-dominant hemisphere → hemineglect.
  • ACA territory: contralateral leg weakness > arm; may produce abulia (loss of motivation/initiative).
  • PCA territory: contralateral homonymous hemianopia, often with macular sparing. Rare: prosopagnosia from fusiform gyrus involvement.

Brainstem syndromes

  • Midbrain: CN III palsy (ptosis, “down and out” eye, dilated pupil).
  • Pons: CN V (facial sensation/mastication) and CN VII (facial expression) deficits; CN VI palsy → inability to abduct.
  • Medulla: CN IX/X/XII deficits → swallowing/voice issues, tongue deviation. Severe medullary lesions threaten respiration.

Cerebellar syndromes

  • Cerebellar lesions cause ipsilateral signs (the pathway double-crosses).
  • Vermis: wide-based, “drunk” gait; trunk ataxia.
  • Hemispheres: ipsilateral intention tremor, dysmetria, dysdiadochokinesia.
  • Flocculonodular lobe: balance and eye movement disturbance.

Spinal cord syndromes

  • Brown-Séquard (hemicord): ipsi motor + vibration loss, contra pain/temp loss 1–2 levels below.
  • ASA infarct: bilateral motor + pain/temp loss; vibration/proprioception spared.
  • PSA infarct: isolated bilateral vibration/proprioception loss.
  • Central cord syndrome: bilateral pain/temp loss with motor + vibration spared.
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Self-assessment · Core Recall

11 board-style MCQs.

Active recall is the highest-yield study method for the INBDE. Pick an answer, check it, and read why every distractor is wrong — that's where the learning compounds.

In the book — different question type

The MCQs above are Core Recall — testing what you've memorized. The book adds a full Clinical Integration set: board-style patient scenarios where you apply this anatomy to real clinical reasoning. That's the section the INBDE actually weights heaviest.

  1. Question 1
    Easy
    A lesion affecting only one half of the spinal cord is called:
  2. Question 2
    Moderate
    In Brown-Séquard syndrome, which function is lost contralaterally below the lesion?
  3. Question 3
    Moderate
    Anterior spinal artery infarct classically spares which sense?
  4. Question 4
    Moderate
    Stroke in which artery is most likely to cause aphasia in a right-handed patient?
  5. Question 5
    Moderate
    Stroke in the anterior cerebral artery (ACA) most often causes:
  6. Question 6
    Easy
    Which stroke syndrome causes contralateral visual field loss with preserved motor strength?
  7. Question 7
    Moderate
    Non-dominant (right, in right-handers) MCA stroke is most likely to cause:
  8. Question 8
    Moderate
    Which cranial nerve deficit is most typical of a midbrain lesion?
  9. Question 9
    Moderate
    Pontine lesions often cause deficits in which cranial nerves?
  10. Question 10
    Easy
    Cerebellar lesions produce deficits on which side of the body?
  11. Question 11
    Easy
    Sudden facial droop and inability to produce speech, but with intact comprehension, suggests:
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